Please use new.greek.doctor

Hello again dear readers!

As work on new.greek.doctor continues, it is getting closer and closer to replacing current greek.doctor completely. Most pages have been copied to the new.greek.doctor, and many of them have been rewritten completely. For this reason, please use new.greek.doctor for those courses that have been copied over already. Also, I won’t make error correction or improvements to this greek.doctor anymore.

new.greek.doctor has a new function too, where it auto-generates PDFs of subjects! Go check it out on new.greek.doctor

Nikolas

greek.doctor’s 6th anniversary and the new.greek.doctor

Hello friends!

Today is this project’s sixth anniversary. If this website was a human it should’ve been able to “bounce ball”, “copy triangle”, and “comfort those in distress”. I thought I’d commemorate this by giving you an update.

Match 1st 2024, only a few days ago, I started specialisation in a speciality which I don’t know how to translate from Norwegian, but I believe it is similar to “emergency medicine” in other countries (akutt- og mottaksmedisin in Norwegian). Up until that date, I was a LIS1, which is an obligatory rotational practice every doctor in Norway must complete before starting specialisation proper. I’m very excited!

I still work on greek.doctor, at least once weekly, by rewriting and copying topics to a new platform on a new website, aptly called new.greek.doctor. You will recognise this platform, called mediawiki, as being the same platform Wikipedia is using. This platform is much better suited for this project than the platform this page is using (WordPress), which is actually a platform for blogs. If only I’d known that from the start so I wouldn’t have to transfer everything now.

Copying pages from here to there is time-consuming (I started doing that more than 18 months ago!) and there is still a long way to go, mostly because I cannot resist rewriting topics when copying them because writing topics is much more fun than copying old topics and ignoring how I know I can do a better job writing them now than when I originally wrote them.

There are many advantages to the new platform. My favorite one is that I can have a “main” or “master” page for a certain disease or subject (like heart failure), and I can then make the topics which are related to that subject extract the parts of that subject which are relevant for the exam in that course (for heart failure that would be pathophysiology, pathology, cardiology, clinical biochemistry, etc.). This allows what I write to be more “centralised” and more easily updatable and reusable across courses, and so that students will recognise material I write from previous courses. It’s also more satisfying for me than to write about the same thing in 5 different topics (probably the most important reason I’m doing this).

I’m also planning to implement a long-awaited feature which generates a pdf file of all the pages for one course, to ease offline use. The pdf will be updated automatically daily or weekly. I could technically implement this on old.greek.doctor (that’s what this will be called from now on) but I won’t bother with that when I’m moving platforms anyway.

Other advantages include:

  • better search function
  • easier to cross-link and navigate between related topics
  • ability to look up subjects without knowing the course it belongs to
  • Students can suggest changes to pages (maybe)
  • the little summary box at the top of the page (takes time to implement)
  • [other advantages I’ve forgotten about]

Because of my full-time work I won’t have a lot of time to dedicate to greek. I’ve also started (paid work) to write for the Great Norwegian Encyclopedia (store norske leksikon), which also takes time. But, greek.doctor is important to me, so I’ll take the time to work on it still. But don’t remain on the edge of your seats waiting for me to finish the migration.

If anyone would pay me to work on greek full time so I wouldn’t have to work clinically, I’d rewrite every single topic and write notes for all subjects I haven’t already written about. If not, there aren’t enough hours in the day to get that done. If anyone is interested, hit me up on email for business inquiries.

The new page is up and running and can be used, but not all topics can be found there yet. Many topics there have been completely rewritten or improved though, which may be of use. Try it!

Until next blog post!

A survey for students’ mental health

A professor at the university of Pécs is conducting a countrywide study, investigating international medical students’ well-being and mental health. The survey is accredited and anonymous.

They have asked for my help in bringing this survey to medical students in Hungary. Please take 12-18 minutes of your time to fill out this survey:

https://forms.gle/dmuv2kVZNjMJrteu9

My honest experience with the university

Spoiler alert: It’s not good.

I’ve spent way too much time writing 21 pages about my experience with the medical university of Pécs, intended to give potential students a look into the life of a student at POTE. You’ll find it in the sidebar.

I also want other students to be able to share their opinion about the university with potential students here. The only thing I require of you is to answer the simple question of “would you recommend this university to a friend or not”. Other than that, you can write whatever you want. Both positive and negative experiences are welcome of course. If you want do to so, message me (email or messenger) with what you want posted and whether you want to be anonymous or not.

My last ever exam experience: the oral state exam

via GIPHY

The oral state exam. The last ever exam at medical school.

It’s not known to be difficult; in fact, it’s known to be more of a formality. Virtually no one fail, and people get good grades despite being unable to answer many of their questions.

Even knowing this, the days before the exams were tough. I wasn’t afraid that I wouldn’t pass, but I was still very stressed. I can’t recall the last time I actually couldn’t eat for days due to stress. But hopefully this was the last time.

How it works

On the day of the written state exam (May 25th for us), all 6th year students receive a schedule for the oral state exam, showing you which day you’ll have the exam on. It’s mostly in alphabetical order, and mine was to be June 1st.

On your designated day of oral state exam, at 7:30 in the morning, a Teams group call begins with officials from the university and all the students who are to have their exams that day. There, the officials randomly draw which department each student is to have their exam at. The students must show up at that department at 8:45, and the exam begins at 9.

At 9 you will get a patient to examine. In some departments, mostly ob/gyn and psych, you’re only to take the patient history and not do any physical examination. Following this, there is a practical part where an examiner asks you questions regarding the case and asks you to show certain examinations. This differs a bit from department to department, as some departments combine this “practical examination” with the theoretical examination which comes next.

The next is the theoretical examination. In each department 4 – 5 examiners from different departments/specialities form a board which will examine you. Some of these examiners may be from other medical universities in Hungary. They’ll ask you a few questions from their own specialities, and often look at your thesis and ask you a few questions about it as well.

Some departments won’t give you your grade until everyone has finished their theoretical examination that day, while some give it right away. After you get your grade (and a small rite of passage from the examiners), you’re free of the tyranny!

My state exam

The uni officials drew psychiatry department for me. I went there at 8:30. At 9 I get a patient. Many patients at the psychiatry clinic speak English, so students having their exams there usually get an English-speaking patient. Initially, they told me that there were no available English-speaking patients, and that I would have to get one who only speaks Hungarian, but that a resident would help me out, but when I got my patient, they did speak English, so I was glad. We sat down and had a nice chat for some time as I tried to take their medical history.

After I was finished, the resident comes and talks to me about the patient (not to examine me). She shows me the patient’s chart and medication list, explains their findings, and answers my questions about the patient. Then, there was nothing to do but wait for the board.

I was the second to last student to enter the room. The board consisted of prof Tamás Tenyi from psych, prof András Tárnok from paeds, prof Imre Szabó from internal medicine gastroenterology, and prof András Papp from general surgery. They ask for my thesis (but not my ID or skillbook, neither of which I didn’t have to show), and they start skimming it while prof Tenyi asks me to talk about the patient.

My patient had psychotic symptoms, so after I was done presenting the patient, he starts asking questions related to this.

  • Tenyi: What is the definition of psychosis?
  • Me: It’s when a person loses contact with reality, and their ability of reality testing.
  • T: Yes. What are the symptoms of psychosis?
  • M: Delusions and hallucinations.
  • T: Yes. And?
  • I thought those were the only two. I try to think of something, but can’t.
  • T: It’s typical in elderly patients.
  • M: Delusions of poverty, that they’re being robbed?
  • T: Yes but you said delusion already. It’s something different.
  • M: Then I don’t know.
  • T: What is it called when an elderly patient doesn’t know where they are?
  • M: Disorientation.
  • T: And is disorientation a symptom of psychosis?
  • In my mind, disorientation is unrelated to psychosis
  • M: No.
  • T: Yes it is. If a patient is in Pécs but thinks they’re in Budapest, they’ve lost contact with reality, so it is psychosis. Right?
  • In retrospect I should’ve guessed that the answer was yes based on where this was going.
  • M: Yes, makes sense.
  • T: Okay. Tell me the eating disorders.
  • M: Anorexia nervosa, bulimia nervosa, orthorexia nervosa, and inverse anorexia nervosa.
  • T: What are the subtypes of anorexia nervosa?
  • M: Binging purging type, and …
  • I couldn’t remember the other type
  • T: Okay. What are the symptoms of bulimia nervosa?
  • M: Binge eating followed by purging, Russel sign on the knuckles, and weight …
  • I pause as I suddenly forget whether bulimia patients are low, normal, or high weight
  • M: … disturbance
  • T: Okay. What is it called when someone is obsessed with healthy eating to the point where they develop malnutrition?
  • M: Inverse anorexia nervosa. I mixed them up
  • T: No, that’s something else. You said it earlier.
  • M: Oh, it’s orthorexia nervosa.
  • T: Yes. So what is inverse anorexia nervosa?
  • M: It’s when a person like a bodybuilder thinks that they’re small.
  • T: Yes. What are the similarities between regular anorexia nervosa and inverse anorexia nervosa?
  • M: Both patients cannot see themselves for what they really are. What they see in the mirror is not the truth.
  • T: And what is this called?
  • M: Body dysmorphia?
  • T: No, body image.
  • What?
  • T: Okay. What is delirium?
  • M: Delirium is an acute decline in sensorium and awareness of the environment caused by somatic disease.
  • T: Yes. What are the most common causes of delirium?
  • M: There can be many causes. Fever, surgery, cancer, infection, metabolic disorders…
  • T: Which are the most common metabolic disorders?
  • M: Electrolyte disturbances, and-
  • T: Which are the electrolyte disturbances most commonly causing delirium?
  • I’d never considered this, but the answer is always sodium, right?
  • M: Sodium disturbance?
  • T: Up or down?
  • M: Hyponatraemia.
  • T: Yes. What else.
  • What else indeed?
  • M: Potassium?
  • T: No.
  • M: Calcium?
  • T: No. It’s not an electrolyte.
  • M: Oh. Hyperglycaemia?
  • T: Can be, but it’s not common.
  • I try to think of other metabolic causes, but I’m drawing a blank.

Sometime around this point in time, I realise that there is a lot of tension in the room. Everyone is just staring at me, not smiling, while I mostly look at the floor. A friend of mine had the exam earlier that day with the same board, and he talked about how chill the atmosphere was and how everyone was laughing. When I realised that my exam had a totally different atmosphere, I start to get a bit anxious. Was there tension because I was doing poorly? Or do they not like me?

  • Tenyi: Think about an elderly patient who is okay in the morning but in the evening they have delirium.
  • That did not help me at all
  • T: Think about heart disease, lung disease, …
  • Still no clue
  • T: … anaemia, …
  • M: Hypoxia?
  • T: Yes!
  • He lets out a small sigh of relief.
  • T: Okay, which other metabolic disorders. It’s something you always check in routine labs.
  • I try to picture routine labs in my mind
  • T: Think about the kidney, liver …
  • M: Oh, of course. Liver and kidney failure.
  • T: Yes. What is liver failure called?
  • M: Cirrhosis?
  • T: Yes, but what is the name of the condition when a patient with liver failure develops neurological and psychiatric symptoms?
  • M: Oh, hepatic encephalopathy.
  • T: Yes. Do you know the name of the tremor?
  • M: Flapping tremor
  • I made a small flapping motion with my hand as I said it, as if my words weren’t enough to convince them that I knew the answer
  • T: Yes. And what is it called for kidney failure?
  • I didn’t know what he was after, but I saw an opportunity for a bad joke
  • M: Renal encephalopathy?
  • They chuckle a bit
  • T: No, it’s uraemia.
  • Duh.

I think that was the questions I got from psych. He says that he’s finished, and gives the word to prof. Tárnok. He introduces himself, and says that he finds my thesis very interesting as he’s an expert on IBD.

  • Tárnok: Do you know what kind of doctor you want to be?
  • Me: I’d like to go into internal medicine, probably nephrology or cardiology.
  • T: Not gastroenterology?
  • M: No, I find nephrology and cardiology more interesting.
  • T: Okay, that’s a shame. And may I ask where you’re from?
  • M: I’m from Norway.
  • T: I looked through your thesis, and it looks very interesting. During researching for your thesis, did you come across the IBSEN study?
  • I pretend to think for a bit, but I’m pretty sure that’s not familiar to me
  • M: No, I haven’t heard of it.
  • T: It is a famous study on IBD which was made in Norway. Anyway. Could you tell me some differences between IBD in childhood and adulthood?
  • This is something I’d never really considered. The only difference I could think of at the time was something I’d read while researching for my thesis
  • M: Well I know that in children total enteral nutrition is used to induce remission in Crohn disease, but this is not used in adults.
  • T: Okay, that is true, but what about the clinical features?
  • M: I don’t know
  • I don’t remember if he gave me the answer or if he just moved on to the next question
  • T: Which is worse, IBD onset in childhood or adulthood?
  • I don’t know why, but for some reason I felt that the counterintuitive answer was more correct
  • M: In adulthood?
  • T: No, childhood IBD is worse. The earlier the onset, the worse the disease.
  • I clearly remember that everyone paused at this point, and this awkward moment was perfect to fuel my already high anxiety
  • T: Can you tell me a bit about the treatment of Crohn disease in childhood?
  • Finally, something I feel like I know
  • M: In children we can use either total enteral nutrition or steroids to induce remission, and we can use immunosuppressants like azathioprine or biologics like adalimumab and infliximab to maintain remission
  • T: Can we use biological drugs to induce remission?
  • M: No, they’re used for maintenance of remission.
  • T: That’s not true, they can be used for induction as well. Can you please tell me which drugs for induction of remission induce mucosal healing?
  • While studying CD I’ve never heard the term mucosal healing, so I had no idea
  • M: Steroids?
  • T: No, the biological drugs. Steroids treat the clinical disease but do not induce mucosal healing, while biological drugs do both. Could you explain to me what step up and top down therapy is?
  • M: Step up is when you begin with the least intensive treatment and gradually increase it, while top down is the opposite.
  • T: Yes, and which do you use in Norway?
  • M: Uhm.. I don’t know.
  • T: Okay, do you know who decides whether the patient gets step up or top down therapy?
  • M: The treating physician?
  • T: No, it’s actually the economists. Top down is more effective but is also more expensive, as biological therapy is more expensive. But, it may pay off in the long run due to better outcomes, so these economists must decide whether the initial increased price is compensated by better outcomes in the long run.
  • I don’t recall his answer exactly, but it was something like this. Definitely was an unexpected question, at least.

I think he had more questions, but I can’t recall them anymore. Eventually he gives the word to prof. Szabó.

  • Szabó: We’ve been talking about IBD so far, which can increase the risk for colorectal cancer. Can you give me some hereditary syndromes which increase the risk for colorectal cancer?
  • Me: Gardner syndrome and familial adenomatous polyposis.
  • Sz: Yes, and there is one more.
  • I try to think, but I can’t recall
  • Sz: It’s a non-polyposis syndrome
  • I didn’t realise how big of a hint this actually was when he said it
  • Sz: It’s hereditary non-polyposis colorectal cancer. Do you know the gene which is mutated in this syndrome?
  • I don’t recall, but I do remember some genes involved in CRC.
  • M: APC?
  • Sz: No.
  • M: P53?
  • Sz: No.
  • Well, those were the ones I could think of initially. But after thinking a bit, I recall something else which is worth guessing
  • M: Microsatellite instability?
  • Sz: *visibly surprised* Yes, MLH1 or MSH2, for example.

He gives the word to the final member of the board, prof. Papp.

  • Papp: I only have one question for you. Could you tell me the differences in surgical treatment for Crohn’s disease and ulcerative colitis?
  • Me: So in UC, surgery can be curative, as it only affects the large bowel, and one can remove all of it, but in CD, surgery can never be curative, as the disease will always recur in the areas next to the excised lesion.
  • P: Well yes, that’s true, but what I was thinking of was that in CD you only remove the diseased segment of the bowel, while in UC you remove the healthy part as well.

And that was the last question. Before I leave, prof. Tárnok asks whether I’m certain that I don’t want to be a gastroenterologist. Before I can answer, prof. Papp says (in a joking, non-malicious way) “no, it’s too difficult for him”, after which everyone laughed a bit. It may sound rude, but I could tell that he didn’t mean it in a rude way. The fact that everyone laughed made me feel a bit better.

I think I was in for 20 – 25 minutes. In most cases, no one gets their grade until everyone has had their theoretical part, and that was the case for my group as well. Luckily, I was the second to last student, so I only had to wait for one student to finish to get to know my grade.

I felt pretty shit before the exam, but I actually felt even more shit after it. I knew that they don’t fail anyone, but I felt like I answered wrongly on almost every question they had, and I had an unshakeable feeling that I’d failed.

After the last student finishes, they call all the students back into the room where we had the exam. Prof. Tenyi opens with a small “rite of passage” where he says that from this day on, we’re no longer students, but that the medical profession requires regular studying to stay up to date, etc etc. This made me feel pretty secure that I’d passed, but he follows it up with “however, the performances have not been equal” or something like that. He starts by reading the grades of everyone in the order they had the exam.

As soon as they say my name and “five”, I immediately burst into tears, in front of everyone. Like, proper ugly crying. The relief was immeasurable. Luckily I was the second to last student, so I only cried while they were reading the result of one more student. As soon as they finish and congratulate everyone, I turn to hug and cry in a friend of mine’s arms who also had the exam that day.

I leave the room, and outside some of my closest and bestest friends are waiting for me with lots of beautiful flowers and bouquets, and so my crying reinforces as the tears are no longer just tears of tiredness and relief, but from being super touched as well. They see me ugly crying and assume the worst (understandably), but I tell them I got a 5, and they become a bit confused as to why I’m crying so much. While outside, still crying, prof. T´´arnok comes over to shake my hand and congratulate me. Just a bit awkward.

Parting words

A select few graduating students had experiences like me, where they were in for a long time and got asked some difficult questions. The vast majority had really simple, 5 – 10 minute exams with baby questions. But, the most important thing is that, no matter which of these two group you end up belonging to, you’ll pass. The oral state exam is not an exame where anyone fails, but it’s still a source of severe anxiety for many, as it’s the last of the last, and failing it would be devestating. Personally, I was super nervous and lost my appetite for almost a whole week before the exam, and it took some days after the exam for these feelings to disappear. Honestly, by having a terrible oral state exam experience, you’ll leave the university with a proper POTE exam as your last exam experience, which is kind of poetic. Full circle.

In the two weeks since the exam, the graduating students have celebrated, by dancing in a public fountain to the music of a live band (a POTE tradition), by going on a trip to lake Balaton, and by partying a lot. We’ve also rested, for the first time in 6+ years without any school-related worries. Even 2 weeks after finishing, we still have moments of realisation, where we realise that we’re actually actually finished. Our Neptun now shows our student status as “Graduated”. From now on, the adult life starts, and a big chapter of our lives is completed, for good and bad.

I’ll write a post later with what’s next for the website. Even though I’m finished, I still have plans to improve and maintain it, and I’ll definitely not take it down, as many have been worried about.

Congratulations to the graduating class of 2022!

Collecting the written state exam questions into one document

The written state exam is May 25th. For those who are unaware, this exam consists of 180 multiple choice questions in 150 minutes, and all the questions are provided upfront on the website finalexam.hu. However, the website only allows viewing 20 questions at a time. Questions may change every year, and so documents from previous years containing all questions are likely not very useful. As such, it’s beneficial to find a method of collecting all questions into a document. Copying and pasting manually takes a lot of time, so I tried to come up with a better solution.

I don’t have a lot of experience with programming, but I knew that I know enough to program a script which automated the process. After a few days of working on it, it’s finished. The repository for the source code is here, and the code will likely continue to work in the future, and so can probably be reused in the future.

I’ve uploaded the document with all the questions to the facebook group for 6th year.

Next, I’ll try to automate the process of converting the questions to flashcards.

Edit: Flashcard conversion is done. The repository is here, and the flashcards have been uploaded to the facebook group.

Edit2: The link to the 2022 state exam questions anki is here. Note that questions may change from year to year, so it would be best if someone remade the anki every year using the tools I made (or with self-made tools).

Internal medicine final exam experience

So I had my internal medicine final at 1st department on the 21st. A notice on Neptun told us to meet at the “Clinical and Educational office” in the B wing of the 2nd floor (room B227) at 8:00, and to bring our 8 case reports and certificate of finishing the practice. When we arrived, they took photocopies of the case reports and certificate, and gave us a paper which we were to bring to the practical part of the exam. We were told to wait outside, and eventually prof. Gabriella Pár comes and gets us and gives a patient each. We were two students per room, each with their separate patient. She tells us that she’ll be back to ask what we’d found in 30 minutes.

Practical part

I introduce myself to the patient and begin taking the anamnesis and performing the physical examination. After me and the other student finish with each of our patients, we leave the room and wait for the professor in the hallway while we collect our thoughts on the patient. While waiting, my patient comes out of the room and offers both of us some tea ❤️.

Eventually, after maybe an hour, the professor arrives. She asks me what I’d found, and I begin presenting my findings. My patient had arrived to the hospital due to abdominal pain which occured after eating. She stops me and begins asking questions.

  • Examiner: What can cause abdominal pain after eating?
  • Me: For example gallstones and gastric ulcer.
  • E: Yes. In case of duodenal ulcer, when does the patient have pain?
  • M: At night
  • E: Yes, and two-three hours after eating. What else can cause abdominal pain after eating?
  • M: Acute pancreatitis.
  • E: Yes. In case of gallstones, where does the pain radiate?
  • M: To the shoulder (thanks to my fellow student who just told me this before my exam, otherwise I might’ve not remembered)
  • E: Which shoulder?
  • M: The right shoulder (I had to think for an embarrassingly long time to recall which side the gallbladder is on)
  • E: And in case of acute pancreatitis?
  • M: It radiates to the back.
  • E: Why does the pain in acute pancreatitis radiate to the back?
  • M: Because they share the same nerve supply? (I don’t remember what I said but it was something vague like this)
  • E: Something like that. What are the risk factors for gallstone?
  • M: It’s the six F’s: female, forty, fat, fair-skinned, fertile, …, and I can’t recall the last one
  • E: Does your patient have any risk factors for gallstones?
  • M: He’s fair-skinned and overweight.
  • E: Anything else?
  • M: (thinking but can’t recall anything)
  • E: What are the types of gallstone?
  • M: Cholesterol and pigment stone… oh, so dyslipidaemia is a risk factor.
  • E: Yes. How can you tell that your patient has dyslipidaemia?
  • M: (I couldn’t see any xanthomas or xantholasmas, so I don’t know)
  • E: It’s a question you asked your patient.
  • M: (What question could I possibly have asked to determine that)
  • She looks at the paper in which I’ve noted my patient findings and points at the medicine list.
  • M: I could ask if my patient takes statins?
  • E: Yes, exactly. Does your patient take statins?
  • M: I’m not sure. He told me that he takes heart medicine but I didn’t ask more specifically. He wanted to show me the list of medications but he couldn’t find it.
  • E: It’s okay. What other question you asked could show that he has dyslipidaemia?
  • I look back down at the paper
  • M: He had a myocardial infarction in the past
  • E: Yes, exactly. What side effects can statins have?
  • M: It can cause myopathy or liver failure, for example.
  • E: What is myopathy?
  • M: Inflammation of the muscle which can cause muscle weakness and pain.
  • E: And what abnormality can you see on the labs?
  • M: Elevation of creatinine kinase.
  • E: Yes. What can be a complication of rhabdomyolysis?
  • M: It can cause kidney failure.
  • E: Good. How much alcohol does your patient drink?
  • M: He said he drinks only a little, but I don’t know how to ask more specifically.
  • E: Okay. What’s the daily limit of alcohol intake?

This is something I wouldn’t have learned unless a friend of mine had gotten the same question and warned me about it, so I looked it up in the lecture.

  • M: It’s 20 – 30 g for men and 10 – 20 g for women.
  • E: Yes. How much alcohol is there in one beer?

I’ve always thought that one beer was one unit, and I’m pretty sure it said on the lecture that one unit is 10 g.

  • M: It’s 10 g.
  • E: No, that’s not correct.
  • M: But it depends on the strength and size of the beer, right?
  • E: Okay, how much alcohol is there in 1 dl of regular beer?
  • M: 3g? (pure guess)
  • E: No, it’s 4 – 5g. So how much in halv a litre?
  • M: 20 – 25g.
  • E: Yes, so one beer is already above the limit for women and above the limit for men. Please show me examination of the liver.

I percuss the border of the liver.

  • M: His liver is 1 finger below the costal arch.
  • E: Okay. What can cause the dull sound over the liver to disappear?

That’s a problem I’d never considered or heard of before, but I figured that the only way it can disappear is if air gets between the abdominal wall and the liver.

  • M: If there is air in the abdomen?
  • E: Yes. How can you diagnose air in the abdomen?
  • M: You could use x-ray or CT.
  • E: How would you see abdominal air on x-ray?
  • M: There would be air under the diaphragm.
  • E: When the patient is in which position?
  • M: When they are standing.
  • E: And if they are lying down?
  • M: Then it would be beneath the abdominal wall?
  • E: Yes. What can cause abdominal air?
  • M: A perforation
  • E: Perforation of what?
  • M: The gastrointestinal tract.
  • E: Yes. What is the treatment for GI perforation?

I was kind of confused, because I know of no internal medicine-related treatment of GI perforation. Could there be an endoscopic procedure? No, there can’t be. It has to be…

  • M: Surgery?
  • E: Yes. So it’s a surgical problem and not an internal medicine problem.

And that was the last of the question on the practical part. She writes a 5 on the paper from earlier, and signs and stamps it (of course). She tells me that my examiner will be prof. Alizadeh and that the theoretical part will take part in the science building in room B001, and that he’s impatient and that I have to hurry there, so I thank my patient and rush over there.

Theoretical part

After rushing over there, I found like 10 students waiting outside the room. They’d also been told to hurry, just to have to wait when they got there. One person already finished their theoretical exam but didn’t get their grade because the examiners had told them that no one will get their grades until all 11 students have finished their theoretical examination. So I sit down to wait. After the 9 students before me have finished, it’s my turn. I go into the room, sit down in front of prof. Alizadeh of haematology and (I think) prof. Tótsimon of cardiology. In my fluster I tell them “good afternoon” even though it was like 10:30. As I sit down, Alizadeh leaves the room, presumably to go to the toilet. Prof. Tótsimon begins the questioning in his absence.

  • Examiner: Tell me about anticoagulants. Which types do you know?
  • M: You have the vitamin K antagonists like warfarin or dicoumarol, the DOACs like dabigatran and apixaban, and the heparins, both unfractionated and LMWH.
  • E: Good. In which cases do we use anticoagulants?
  • M: Anticoagulants are used in case of atrial fibrillation to prevent stroke, in DVT/PE, and in acute myocardial infarction.
  • E: Good. There’s one more.

I try to think but I can’t recall it.

  • E: It’s also used in nephrotic syndrome, but it’s okay if you don’t know. (I actually did know but I’d never thought of that in a million years)
  • E: When do we use vitamin K antagonists and when do we use DOACs?
  • M: Generally, VKAs are only indicated in case of prosthetic heart valves or in case of moderate or severe mitral stenosis.
  • E: Good. In which case of atrial fibrillation do we use anticoagulation?
  • M: If the CHA2DS2-VASc score is 1 in men or 2 in women.
  • E: Actually in case of 1 for men or 2 for women, anticoagulation can be considered, but if it’s 2 in men or 3 in women, then it’s recommended. But it’s good, your answer is good. Do you remember the parameters of the CHA2DS2-VASc score?

This is something I imagined could be a question, so I’d prepared for it. I close my eyes and try to visualise the mnemonic. To my own surprise, I recite it correctly.

At this point prof. Alizadeh returns to the room. Prof Tótsimon quickly explains him in Hungarian what we’ve talked about so far. Prof. Alizadeh continues:

  • E: So my colleague tells me that you’ve talked about anticoagulants and that you’ve answered excellently so far. Now, please tell me. What are the antidotes for DOACs?

I was not prepared for such a pharma question. I remember studying it in pharma but I hadn’t looked at it since. We spend some time in silence as I try to dig deep into my memory for the information, but I can’t recall it.

  • E: Okay then please tell me, what are different types of DOACs?
  • M: You have dabigatran, which inhibits factor II and X, and the others like apixaban and rivaroxaban, which inhibit only factor X.
  • E: Dabigatran inhibits only factor II, but yes. Now, each of these groups have different antidotes. Please tell me what they are. The antidote for dabigatran begins with an “I”.

The hint made some gears turn in my head.

  • M: Is it something like idarocumab? (I don’t recall exactly what I said, but it was something like this, which was not very close to the correct name)
  • E: Yes, it’s idarucizumab. The antidote for the others are andexanet alpha and ciraparantag. Now, for a better grade, please tell me the newest drug for atherosclerotic ischaemic heart disease.

At first, I’m like, what. I’m pretty up to date on cardiology, but I can’t recall any new drugs for that indication. I know that SGLT2 inhibitors were recently indicated for heart failure as well. Could that be what he meant? But that’s not related to atherosclerosis.

  • E: It’s four letters and a number. It’s an injectable drug.

Okay, but then it has to be them, right?

  • M: SGLT2 inhibitors?
  • E: No, that’s for heart failure.

I knew it.

For the next part, I believe they gave me a hint which put me on the right track to the answer, but I can’t recall what the hint was. It might’ve been the first letter, “P”.

  • M: It’s PGSL2 inhibitors, or something (I don’t recall exactly what I said, but it was nowhere close to the correct answer lol)
  • E: Yes, it’s PCSK9 inhibitors. It’s okay, please go out and wait for your grade.

Epilogue

After waiting for the 11th student to finish their exam, everyone went back into the room one at a time to get their grades. As I enter when it’s my turn, the examiners are already on their way out, so I think they kind of forget about me. As they walk past me, they tell me that I got “excellent” and congratulated me.

And that was it! That was my last ever exam at POTE. Now only state exams remain. I was surprised to be asked such an amount of pharma questions, but anyway.

Neurology final exam experience

So I had my neurology final yesterday. The exam was to begin at 10:30 (which sucks, I prefer having early exams). I was waiting outside the neurology department (one floor up from psychiatry) with the other students. According to Neptun, our examiner was to be prof. Janszky.

It’s 10:30. A doctor comes and guides us to the department’s “library”, quotation marks because there were zero books in there..

Practical part

She asks who of us had reflex hammers, which only I and a few other students had brought (but I’d forgotten my lab coat, so we were even). Those of us who had forgotten to bring lab coats were given disposable isolation gowns, and those who’d forgotten to bring a reflex hammer were either lent one or told to borrow from those who had one.

It’s 10:46. The doctor gives each of us a patient to examine, and tells us that we should perform a full neurological examination, and after 20 minutes, she’d return and ask what we’d found and ask a few questions.

So we do the examinations on our patients, and then we wait. The wait turns out to be more like one hour. (She had to examine four students before me, which is why it took so long.)

When she finally returns, it’s 11:38. She begins by asking me what I’d found. She then asks a few theoretical questions:

  • What is intention tremor?
  • Where can the lesion be if there is an intention tremor?
  • How do you examine the muscle tone?
  • What is rigidity and what is spasticity?
  • If there is a lesion in the pyramidal tract, what kind of muscle tone can be present?
  • Demonstrate Hoffman and Trömner sign
  • What is the other major pyramidal sign and how do you examine it?
  • What are the signs of peripheral paresis?
  • Demonstrate on the patient how you would examine the visual field.
  • What’s hemianopia?
  • Let’s say that I had a stroke in my right hemisphere. What kind of visual field disturbance could I experience?

And that was it for the practical part. I didn’t answer all questions perfectly, but more or less, so I got 5 in the end. It took five minutes.

Theoretical part

She tells me to go back to the library to draw topics. It’s 11:45, lunch time. I draw my three topics:

  • A/9: Muscle biopsy
  • B/29: Myasthenia gravis
  • C/22: Treatment of traumatic brain injuries

My B topic was great, C was okay, and A was not the best. I sit down to write my topics, but there are three students before me, plus the student who was currently having the exam. I knew it was going to be a long day, but I hadn’t brought food.

Time passes, but feels like it’s standing still. Each student’s exam takes like 20 minutes. The exam is happening in another room, so we have no idea how it’s going with the examinees. Hunger grows stronger and stronger. I look at the other students, and they ask me what the time is, as I had my watch. “12:30”, I say, and one student rubs their belly to signal that they’re really hungry. I know, I know. I am too.

I’m hungry, restless, and anxious as we’re sitting there waiting. Nothing is happening in the room; everyone’s is just waiting. The doctor watching over us is droodling on a piece of paper. I look outside the window, hoping to find something interesting to look at to pass the time, but the view consists of an opposing wall with flaking paint. My akathisia is getting to strong; I have to move. I get up and start walking back and fourth in the room. My watch counts 1149 steps as I keep walking to pass the time and ease my anxiety.

Finally, the last student before me returns from her exam. I look at my watch; it’s now 13:03. She walks me to prof. Janszky’s office. A nice office. The doctor who examined my practical part is present as the co-examiner. I sit down in a comfy chair, and he tell me that I can start with whichever topic I would like. Nice.

B/29 Myasthenia gravis

I began by mentioning everything I’d written down, except the part about Lambert-Eaton. I wasn’t sure if I should mention the treatment, but I did anyway and he didn’t correct me or stop me.

He asks me whether there are any other antibodies associated with MG. I forget about anti-MuSK so I mention the anti-VDCC antibodies, but he replies that they’re associated with Lambert-Eaton and not MG. I draw a complete blank on the anti-MuSK. After a while, he gives me the answer, and tells me to go to the next topic.

C/22 Treatment of traumatic brain injury

I begin with who needs a CT and who don’t. It’s probably not actually a part of the topic, but I found it interesting so I wanted to include it anyway. I open with “So I’m Norwegian, so I’ve looked at the Scandinavian guidelines” they laugh a bit and say something like “maybe they’re better”.

After explaining that, I begin by talking about how we only can treat secondary injuries and not primary, etc. etc. At some point he stops me to ask about the types of TBI. He begins with commotion (which I think is a Hungarian term for concussion), and asks me to list the others. I explain contusion, ICH, SAH, SDH, and EDH, and say that if there’s a haematoma we can drain it and if there’s bleeding in the intraventricular space, we can use a shunt. He agrees. Afterward I continue with the supportive treatments I’d listed for oedema, vasospasm, etc.

He asks me what the lucid interval is, and I answer. He asks me why we examine the eyes in TBI, and I say it’s because anisocoria could be present. He asks why anisocoria occurs, and I say herniation of the cerebral peduncles, to which he replies that it’s wrong, it’s rather uncal herniation. “In case of anisocoria, which pupil is on the side of the injury?”, to which I reply “the larger pupil”. He agrees.

He asks what another finding may be on the eyes in case of TBI. “Fixed pupil, when there’s no reaction to light”, I respond. He asks which nerve is affected in this case, and I say occulomotor. He signals for me to proceed to the last topic.

A/9 Muscle biopsy

I begin by listing what I’d written down. I don’t get further than the IBM point until he interrupts me and tells me that we’re finished and that it’s a 5. Yay! The exam took 7 – 8 minutes.

On my way out, they ask me for the three envelopes each of my topics were inside when I drew them. I had forgotten them in the library, so I had to go and get them. Awkward!

I look at my watch. It’s 13:13.

Time for lunch.